• Mitochondrial Dysfunction in Autism Spectrum Disorders: A Systematic Review and Meta-Analysis
• Mitochondrial dysfunction in the gastrointestinal mucosa of children with autism: A blinded case-control study
• Mitochondrial Dysfunction Is Inducible in Lymphoblastoid Cell Lines From Children With Autism and May Involve the TORC1 Pathway
• Mitochondrial Dysfunction may explain symptom variation in Phelan-McDermid Syndrome
• Mitochondrial Fatty Acid β-Oxidation and Resveratrol Effect in Fibroblasts from Patients with Autism Spectrum Disorder
• Mitochondrial morphology is associated with respiratory chain uncoupling in autism spectrum disorder
• Modern Biomarkers for Autism Spectrum Disorder: Future Directions
• Modulation of Immunological Pathways in Autistic and Neurotypical Lymphoblastoid Cell Lines by the Enteric Microbiome Metabolite Propionic Acid
• Modulation of mitochondrial function by the microbiome metabolite propionic acid in autism and control cell lines
• Multichannel least-squares linear regression provides a fast, accurate, unbiased and robust estimation of Granger causality for neurophysiological data
• Multivariate techniques enable a biochemical classification of children with autism spectrum disorder versus typically-developing peers: A comparison and validation study
• Network‐based analysis reveals stronger local diffusion‐based connectivity and different correlations with oral language skills in brains of children with high functioning autism spectrum disorders
• Neuropathological Mechanisms of Seizures in Autism Spectrum Disorder
• Novel biomarkers of metabolic dysfunction is autism spectrum disorder: potential for biological diagnostic markers
• Nuclear Peroxisome Proliferator-Activated Receptors (PPARs) as Therapeutic Targets of Resveratrol for Autism Spectrum Disorder
• Objective Phonological and Subjective Perceptual Characteristics of Syllables Modulate Spatiotemporal Patterns of Superior Temporal Gyrus Activity
• Oxidative Stress Challenge Uncovers Trichloroacetaldehyde Hydrate-Induced Mitoplasticity in Autistic and Control Lymphoblastoid Cell Lines
• Oxidative Stress Challenge Uncovers Trichloroacetaldehyde Hydrate-Induced Mitoplasticity in Autistic and Control Lymphoblastoid Cell Lines
• Oxidative stress induces mitochondrial dysfunction in a subset of autism lymphoblastoid cell lines in a well-matched case control cohort
• Oxidative stress induces mitochondrial dysfunction in a subset of autism lymphoblastoid cell lines in a well-matched case control cohort.